Chronic aggressive periodontitis

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What is the cause of periodontitis?

Both chronic and aggressive periodontitis share major environmental and genetic risk factors, and some clinicians find the differential diagnosis between these two diseases difficult and impossible. Smoking, oral hygiene, and psychological factors seem to play a role in both chronic and aggressive periodontitis. What are chronic and aggressive periodontitis ? Chronic periodontitis is an inflammatory process that affects the protective and supportive tissues around the teeth. The primary etiology of this disease is the bacterial plaque on the tooth surface that leads to marginal tissue inflammation, known as gingivitis.

The prevalence of LAP is less than and that of GAP is 0. In aggressive periodontitis , attachment loss occurs at a much faster rate than in chronic periodontitis , the more common variant.

Because the course of destruction occurs rapidly, evaluation of historical, radiographic and clinical data is necessary to estimate the start of disease. Necrotizing periodontal disease, which is the death of the gums, tooth ligaments, and bones due to lack of blood. Aggressive periodontitis , which is hereditary and causes rapid loss of bone and teeth if untreated. The disease is characterized by inflammation of the supporting structures of the teeth and loss of gum attachment due to destruction of the periodontal tissues. Prevalence and severity of the disease increase with age.

This type is caused by plaque buildup and involves slow deterioration that may improve and get worse over time but causes destruction in the gums and bone and loss of teeth if not treated. AgP classified into two categories named localized and generalized aggressive periodontitis. It differs from chronic periodontitis (CP) depending on age of onset of the disease, rate of progression of the disease, structure and.

Depending on the time of diagnosis and the intensity of the disease, the treatment will vary accordingly. Systemic diseases like hematologic disorders and some genetic disorders also show periodontitis as a manifestation mimicking generalized aggressive periodontitis which can be ruled out by assessing the. It progresses at a slow to moderate rate. Bacterial infection resulting in inflammation of the supporting tissues of teeth where this is CT attachment, PL and alveolar and supporting bone loss. Its chronicity is the result of the persistence of the inflammatory stimulus, the plaque biofilm, and the chronic inflammatory infiltrate in the marginal periodontal tissues in response to this.

Only chronic and aggressive forms of periodontitis will be considered for this review owing to different pathophysiology and optimum justification for antibiotic prescription in these other mentioned types of periodontitis. Host response plays an essential role in pathogenesis of chronic periodontits.

EPIDEMIOLOGY Considerably less epidemiological data are avail- able on AgP than on chronic periodontitis. Bartłomiej Górski, Stanisław Jalowski, Renata Górska and Maciej Zaremba, Treatment of intrabony defects with modified perforated membranes in aggressive periodontitis : subtraction radiography outcomes, prognostic variables, and patient morbidity, Clinical Oral Investigations, 10. Acupuncture, Aripiprazole, Cannabidiol, Celecoxib, and Clonazepam to treat their chronic periodontitis and its symptoms. T or F: Refractory chronic periodontitis occurs despite good self-care and appropriate therapy and regular maintenance visits.

Research and findings are presented for two methods of therapy for periodontitis. One is a prospective, randomized clinical study that addresses the treatment of aggressive periodontitis with antimicrobial photodynamic therapy or system antibiotics. Genetic and environmental risk factors for chronic periodontitis and aggressive periodontitis. Can presence or absence of periodontal pathogens distinguish between subjects with chronic and aggressive periodontitis ? The plaque contains bacteria that spreads and infects the surrounding tissue and bone that support the teeth which may eventually lead to tooth loss.

Overall, while most clinicians would agree that aggressive forms of periodontitis exist as clinical entities, the clinical distinction between chronic and aggressive periodontitis (especially generalized) is not clear cut. The overlapping areas represent the number of OTUs shared by the counterpart samples. The major forms of periodontitis , chronic (CP) and aggressive (AgP), do not display sufficiently distinct histopathological characteristics or microbiological. Twenty-one subgingival plaque samples were collected from three patients with chronic periodontitis (ChP), three patients with aggressive periodontitis (AgP) and three periodontally healthy subjects (PH).

Each patient with periodontitis was sampled at three sites, at different probing depths (PDs, one each at mm, 5–mm, and ≥ mm). Slowly progressing disease. Progress aggressively in patients having diabetes, smoking, stress and thyroid conditions.

Common features include rapid attachment loss and bone destruction and familial aggregation.